The American immunologist Dr. William E. Paul has received the Max Delbrück Medal in Berlin. The jury honored the researcher for his work on a key regulator of the immune system, interleukin 4 (IL-4). Dr. Paul works at the National Institute of Allergy and Infectious Diseases (NIAID) at the National Institutes of Health (NIH) in Bethesda, Maryland, USA. Professor Andreas Radbruch, scientific director of the German Rheuma Research Center Berlin, held the laudatory address.
How Bleach Kills Bacteria
Developed more than 200 years ago and found in households around the world, chlorine bleach is among the most widely used disinfectants, yet scientists never have understood exactly how the familiar product kills bacteria. New research from the University of Michigan, however, reveals key details in the process by which bleach works its antimicrobial magic. In a study published in the Nov.
New Ways To Study Development And Disease Following Breakthrough In Cell-Type Analysis
Like skilled assassins, many diseases seem to know exactly what types of cells to attack. While decimating one cadre of cells, diseases will inexplicably spare a seemingly identical group of neighbors. What makes cells vulnerable or not depends largely on the kinds and amounts of proteins they produce - their "translational profile," in the lingo of molecular biology.
LSUHSC Research Identifies Key Contributor To Alzheimer's Disease Process
Walter J. Lukiw, PhD, Associate Professor of Neuroscience and Ophthalmology at LSU Health Sciences Center New Orleans, is the lead author of a paper identifying, for the first time, a specific function of a fragment of ribonucleic acid (RNA), once thought to be no more than a byproduct, in regulating inflammation and the development of Alzheimer's disease.
Alzheimer's Gene Slows Brain's Ability To Export Toxic Protein
The only known genetic risk factor for Alzheimer's disease slows down the brain's ability to export a toxic protein known as amyloid-beta that is central to the damage the disease causes, scientists have found.
The research, published by the Journal of Clinical Investigation, provides new clues into the workings of a protein known as apolipoprotein E4, or ApoE4. People who carry two copies of the gene have roughly eight to 10 times the risk of getting Alzheimer's disease than people who do not.
